Complex karyotype in de novo acute myeloid leukemia: typical and atypical subtypes differ molecularly and clinically
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Citation:
Leukemia vol 33 (7) 1620-1634
Year:
2019
Type:
Manuscript
Funding:
NCTN
Endpoint:
Secondary-not-in-original
Analysis:
Primary
Data Sharing:
No-Data-Sharing
Status:
Presented/Published
Citation Status:
pmc-release
Note:
Methodological:
No
Biospecimen:
Yes
SDC:
No
Book Volume:
8
Parents:
2781  
Children:
None
Program:
TRP
Primary Committee:
Leuk Corr Sci
Sec. Committees:
   
Pharmas:
 
Grants:
U10CA180821, U10CA180882, P30CA016058, U10CA180833, U10CA180850, U10CA180861, U10CA180866, U10CA180867, U24CA196171  
Corr. Author:
 
Authors:
                                   
Networks:
CT063, LAPS-AL002, LAPS-MA036, LAPS-MO011, LAPS-NY158, LAPS-OH007, NC002, NORTHWELL, OH070   
Study
Multiple Studies, or Legacy Studies in Alliance Study:
CALGB-8461, CALGB-9665, CALGB-20202
Phases:
N/A
Keywords:
acute myeloid leukemia, complex karyotype, next-generation sequencing, gene mutations, clinical outcome