FLT3 D835/I836 mutations are associated with poor disease-free survival and a distinct gene-expression signature among younger adults with de novo cytogenetically normal acute myeloid leukemia lacking FLT3 internal tandem duplications
- Citation:
- Blood vol 111 (3) 1552-1559
- Year:
- 2008
- Type:
- Manuscript
- Funding:
- NCTN
- Endpoint:
- Secondary
- Analysis:
- Primary
- Data Sharing:
- No-Data-Sharing
- Status:
- Presented/Published
- Citation Status:
- pmc-release
- Note:
- Methodological:
- No
- Biospecimen:
- Yes
- SDC:
- No
- Book Volume:
- Parents:
- 373
- Children:
- None
- Program:
- TRP
- Primary Committee:
- Leuk Corr Sci
- Sec. Committees:
- Pharmas:
- Grants:
- R01 CA102031, CA102031, CA101140, U10 CA077658, CA098933, CA016058, CA089341, R01 CA089341, CA077658, U24 CA114725, U10 CA101140, U10 CA041287, R01 CA098933, K01 CA096887, P30 CA016058, CA041287, CA114725, CA096887
- Corr. Author:
- Authors:
- Susan P. Whitman Amy S. Ruppert Michael D. Radmacher Krzysztof Mrózek Peter Paschka Christian Langer Claudia D. Baldus Jing Wen Frederick Racke Bayard L. Powell Jonathan E. Kolitz Richard A. Larson Michael A. Caligiuri Guido Marcucci Clara D. Bloomfield
- Networks:
- Study
- Multiple Studies, or Legacy Studies in Alliance Study:
- CALGB-19808, CALGB-20502, CALGB-8461, CALGB-9621, CALGB-9665
- Phases:
- 1, 3, N/A
- Keywords:
- Neoplasia